related to this: https://forums.zotero.org/discussion/67735/a-new-custom-translator-isnt-called-when-visiting-a-target-web-page

SfN Abstract.js

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					// "*M. J. REDINBAUGH1, J. M. PHILLIPS1, N. A. KAMBI1, S. MOHANTA1, A. RAZ2,3, Y. B. SAALMANN1"
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	// 	<span class="title color-primary"><i class="icon-caret-right"></i> <span class="bodyTitle">751.03. Stimulation of central lateral thalamus restores conscious processing under propofol and isoflurane anesthesia</span></span>
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//     751.03
//      / D49
//      - Stimulation of central lateral thalamus restores conscious processing under propofol and isoflurane anesthesia
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//     <dl>
//         <dt>Authors</dt>
//         <dd><b>*M. J. REDINBAUGH</b><sup>1</sup>, J. M. PHILLIPS<sup>1</sup>, N. A. KAMBI<sup>1</sup>, S. MOHANTA<sup>1</sup>, A. RAZ<sup>2</sup><sup>,3</sup>, Y. B. SAALMANN<sup>1</sup>; <br><sup>2</sup>Anesthesiol., <sup>1</sup>UW Madison, Madison, WI; <sup>3</sup>Anesthesiol., Rambam Hlth. Care Campus, Haifa, Israel</dd>
//
//         <dt>Disclosures</dt>
//         <dd>&nbsp;<b>M.J. Redinbaugh:</b> None.&nbsp;<b>J.M. Phillips:</b> None.&nbsp;<b>N.A. Kambi:</b> None.&nbsp;<b>S. Mohanta:</b> None.&nbsp;<b>A. Raz:</b> None.&nbsp;<b>Y.B. Saalmann:</b> None.</dd>
//
//         <dt>Abstract</dt>
//         <dd>The neural correlates of consciousness (NCC) are the underlying brain mechanisms needed to produce a conscious state. Current evidence suggests that frontal-parietal and thalamo-cortical circuits may contribute to the NCC. The intralaminar thalamus, specifically the central lateral nucleus (CL), is reciprocally connected with both frontal and parietal cortices. Damage to the intralaminar thalamus has been linked to disorders of consciousness such as spatial neglect and coma. We hypothesized that CL could promote consciousness by regulating information transmission across fronto-parietal cortex. To test this hypothesis, we contrasted thalamo-cortical activity between conscious and anesthetized states. We also electrically stimulated CL to establish its casual role in regulating fronto-parietal activity and the level of consciousness.<br>We performed simultaneous recordings in the frontal eye field (FEF), lateral intraparietal cortex (LIP), caudate nucleus (CN), and CL in two macaques during general anesthesia (propofol or isoflurane) and wakefulness (resting state or fixation task). We used laminar probes to record spikes and local field potentials (LFPs), and imaged probes in situ to confirm recording sites. We electrically stimulated thalamic sites with 100-300 microA current at 2, 10 or 50Hz. We analyzed data during stable eye epochs at “baseline”, in a passive auditory oddball task, and with thalamic stimulation. To monitor the level of consciousness, we recorded EEG, tracked eye position, and monitored vital signs. Awake and anesthetized recordings took place with the animal’s head stabilized in a dark room.<br>Preliminary results suggest that CL stimulation counteracted the effects of propofol and isoflurane. Stimulating CL at 50Hz during anesthesia increased behavioral measures of arousal (including eye opening and purposeful movements), reduced EEG power at low frequencies and increased EEG power at higher frequencies, as well as enhanced responsiveness to auditory oddball stimuli. These behavioral and neural changes started soon after, and were maintained during, CL stimulation. Stimulation was primarily effective in CL, and not nearby thalamic nuclei. Further, current source density analyses suggest that anesthesia differentially influenced feedforward and feedback processing across fronto-parietal cortex.<br>Our data suggest that CL regulates information processing in fronto-parietal cortex, and thereby the level of consciousness. This not only supports the idea that CL and fronto-parietal cortex contribute to the NCC, but also suggests that propofol and isoflurane may (partly) share systems-level mechanisms of anesthesia.</dd>
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// <span class="session-date"><i class="icon-calendar"></i> November 15, 2017, 3:00 - 4:00 PM</span>